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认为精神分裂症是前列腺素(PGS)缺乏性疾患的根据是:①抗精神分裂症药物兴奋催乳素分泌,由此兴奋PGS分泌:②用PGS对抗剂治疗的病人出现类精神分裂症综合征的现象;③精神分裂症患者具有抗疼痛、炎症和炎症疾患的事实。一、炎症、疼痛与精神分裂症的关系: 已证明,在关节炎、其它炎症反应及疼痛中,PGS起了关键的作用。如果精神分裂症患者伴有PGS缺乏,那么这些患者对炎症的反应性和易感性必然降低。例如澳大利亚报道,301例女性精神分裂症中,无一例见风湿性关节炎的证据。精神分裂症患者有对抗注射组织胺和伤寒菌苗的致炎作用和全身反应,且疼阈增加,疼痛所致的血压和瞳孔反射亦降低,外科或烧伤均不易引起休克。PGS的血管抑制
Schizophrenia is considered to be a prostaglandin (PGS) deficiency disorder based on the following: ① anti-schizophrenia drugs excited prolactin secretion, thereby stimulating PGS secretion: ② patients treated with PGS antagonist schizophrenia syndrome Phenomenon; ③ schizophrenia patients with anti-pain, inflammation and inflammation of the fact. First, the relationship between inflammation, pain and schizophrenia: PGS has been shown to play a key role in arthritis, other inflammatory reactions and pain. If schizophrenia suffers from PGS deficiency, the response and susceptibility of these patients to inflammation are bound to decrease. For example, in Australia, none of the 301 women with schizophrenia reported evidence of rheumatoid arthritis. Patients with schizophrenia have anti-inflammatory effects and systemic reactions against histamine and typhoid bacillus vaccine injection, and increased pain threshold, pain-induced blood pressure and pupil reflexes are also reduced, surgery or burns are not easy to cause shock. Vascular inhibition of PGS