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用分别补充100ppm VE 的大骨节病区低硒粮配制的低硒饲料和加饲料饲硒养并繁殖大鼠,组织学上未发现母代或子代大鼠有肝坏死和骨骼肌病变。在母代大鼠中,低硒组的血硒含量明显地低于加硒组(P<0.01),而血清 VE 含量在低硒与加硒组间几乎相同。提示低硒不影响母代大鼠的血清 VE 含量。在子代大鼠中,各组动物血硒含量的变化与其母代基本相同;而低硒组的血清 VE 含量明显地高于加硒组(P<0.05),并且血清 VE 含量与血硒含量之间呈负相关关系(r=-0.658,P<0.05)。结果表明饲料低硒能使子代低硒大鼠血清 VE 含量增加。
The rats were fed with low selenium diet supplemented with low selenium diet supplemented with 100ppm VE and feed supplemented with selenium, and no hepatic necrosis and skeletal muscle lesions were found in the tissues of the mother or offspring. In the mother rats, selenium content in the low selenium group was significantly lower than that in the selenium group (P <0.01), while the serum VE content was almost the same between the low selenium group and the selenium group. Suggest that low selenium does not affect the serum VE content of the mother rats. In offspring rats, the content of selenium in blood of each group changed basically the same as that of their sellers; however, the serum VE content of selenium group was significantly higher than that of selenium group (P <0.05), and the serum VE content and blood selenium content (R = -0.658, P <0.05). The results showed that dietary low selenium could increase serum VE in offspring selenium rats.