盆腔炎致病推理的经典理论早在1898年 Kelly等在教科书中提到,有二个途径:一、从宫颈开始感染,扩散到子宫内膜表面;二、淋巴引流。其最大的缺点是缺乏感染原引起盆腔炎的发病机理——如何从下生殖道上升到上生殖道。本文提供以下三个致病作用机理。一、滴虫作传染媒介:活动的滴虫携带各种感染原从阴道上升至输卵管,因此可考虑为妇女重要疾病的带菌者。作者见解(一)滴虫能上升到输卵管水平(甚至到腹腔);(二)细菌密切附着在滴虫上。二、精子作带菌者:认为微生物到输卵管是由粘附在精子上运输。有丰富的证据提供精子与各种感染原的亲密关系,认为精子带着作为女性中致 Classical theories of pelvic inflammatory disease reasoning as early as 1898 Kelly et al. Mentioned in the textbook, there are two ways: First, from the cervix began to infection, spread to the endometrial surface; Second, lymphatic drainage. Its biggest drawback is the lack of pathogenesis of the original causes of pelvic inflammatory disease - how to rise from the lower genital tract to the upper genital tract. This article provides the following three pathogenic mechanisms. First, trichomoniasis vector: Trichomoniasis activities carried a variety of original infection from the vagina to the fallopian tubes, it can be considered as a carrier of important diseases for women. The author’s opinion (a) trichomoniasis can rise to the fallopian tube level (or even to the abdominal cavity); (b) bacteria attached to the trichomoniasis. Second, sperm as carriers: that microorganisms to the fallopian tube is attached to the transport of sperm. There is abundant evidence to provide the intimacy between sperm and various infectious agents,