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齐×,女,27岁。二年前,无明显诱因,右手无力,逐渐出现肌肉萎缩,某医院诊断为运动神经元性疾病,用维生素 B_(12)、ATP 及变构蛇神经毒素治疗无效,且渐进性发展。1990年2月26日入院。查体:脑神经无示位征。全身感觉正常。右手骨间肌、大小鱼际肌萎缩,呈“鹰爪”样,右手指肌力Ⅱ级、无握力。右前臂肌肉萎缩,肌力Ⅲ级,肌张力低下,腕上12cm 处臂围16.5cm。左手无肌肉萎缩,指力正常,握力28Kg。左前臂无肌肉萎缩,肌力及肌张力正常,腕上12cm 处臂围21.8cm。右肱二、三头肌腱反射消失,左侧正常。外院肌电检查:符合运动神经元性损害。MRI 检查:C_2～T_2脊髓节段内见低密度
Qi ×, female, 27 years old. Two years ago, there were no obvious causes, right hand weakness and muscle atrophy. A hospital diagnosed as motor neuron disease was ineffective and progressive development with vitamin B 12, ATP and allosteric serotonergic toxin. February 26, 1990 admitted to hospital. Physical examination: no signs of cranial nerve sign. The whole body feels normal. Right hand interosseous muscle, the size of the atresia muscle atrophy, was “Eagle Claw” like, right hand muscle strength Ⅱ level, no grip. Right forearm muscle atrophy, muscle strength grade III, low muscle tension, arm wrist 12cm at 16.5cm. No muscle left atrophy, normal finger force, grip 28Kg. Left forearm without muscle atrophy, muscle strength and normal muscle tension, wrist 12cm arm circumference 21.8cm. Right brachial, triceps tendon reflex disappeared, the left normal. Outer hospital EMG examination: consistent with motor neuron damage. MRI examination: C_2 ~ T_2 spinal cord segments see low density