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疟疾病人贫血,是否完全由于原虫在红细胞内的寄生和破坏作用所致?鉴于黑水热患者在血中疟原虫为数极少的情况下,可以突然发生严重贫血,因而使人想到:疟疾病人的贫血,必尚有其它机制参与。为阐明此种机制,作者选择所在医院热带病科住院患者8名(恶性疟5例、间日疟2例、卵形疟1例)进行研究。~(51)Cr半衰期测定表明:疟疾病人在抗疟治疗结束后,显著的溶血性贫血仍将持续4～5周。感染严重者(如恶性疟),溶血性改变亦最重。但即使是病情轻微得多的问日疟和卵形疟,亦有一定程度溶血。感染期间和治后一定时间内,红细胞表面有补体存在,提示其溶血性改变尚与含补体的免疫复合物有关。没有
Is anemia of malaria completely due to the parasitic and destructive effects of protozoa in erythrocytes? Given that severe episodes of anemia can occur abruptly in a very small number of blood-borne malaria parasites, it is tempting to think of malaria patients Anemia, there must be other mechanisms involved. To clarify this mechanism, the authors selected 8 inpatients (5 cases of Plasmodium falciparum, 2 cases of Plasmodium vivax and 1 case of oval-shaped malaria) in the hospital’s tropical medicine department. ~ (51) Cr half-life measurement showed that: After the antimalarial treatment of malaria patients, significant hemolytic anemia will continue for 4 to 5 weeks. Severe infections (such as falciparum malaria), hemolytic changes are also the most important. But even with slightly milder conditions vivax malaria and oval malaria, there is some degree of hemolysis. During infection and within a certain period of time after treatment, the presence of complement on the surface of erythrocytes suggests that the hemolytic changes are still related to complement-containing immune complexes. No