多囊卵巢综合征(polycystic ovarian syndrome,PCOS)是一种以稀发排卵或无排卵、多毛、肥胖和胰岛素抵抗为主要临床表现的妇科内分泌疾病,其发病率约占育龄妇女的5%~10%[1]。PCOS的病理生理机制主要包括胰岛素抵抗、高雄激素血症、神经内分泌异常。王引凤等[2]临床研究认为胰岛素抵抗可促进PCOS患者肥胖,尤其是腹型肥胖的发生,并与PCOS糖 Polycystic ovarian syndrome (PCOS) is a gynecological endocrine disease with sparse ovulation or anovulation, hirsutism, obesity and insulin resistance as the main clinical manifestations. The incidence of PCOS is about 5% -10 %[1]. The pathophysiology of PCOS mainly includes insulin resistance, hyperandrogenism and neuroendocrine disorders. Wang Yinfeng et al [2] Clinical studies suggest that insulin resistance can promote obesity in PCOS patients, especially abdominal obesity, and with PCOS sugar