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越来越多的研究显示,慢性、亚临床性及非特异性炎症状态与2型糖尿病(T2DM)的大血管并发症发生发展有密切关系。血管内皮细胞及平滑肌细胞分泌的白介素-6(IL-6)在炎性反应中起核心调节作用[1],其可直接作用于血管壁而引起血管壁损伤,参与动脉粥样硬化(AS)的形成。本文对IL-6与T2
A growing number of studies have shown that chronic, subclinical and nonspecific inflammatory conditions are closely related to the development of macrovascular complications of type 2 diabetes mellitus (T2DM). Interleukin-6 (IL-6) secreted by vascular endothelial cells and smooth muscle cells plays a central regulatory role in the inflammatory response [1], which can directly act on the vascular wall to cause damage to the vascular wall and participate in the development of atherosclerosis (AS) Formation. This article on the IL-6 and T2