论文部分内容阅读
已知胎儿宫内生长迟缓(IUGR)与胎盘梗阻有关。Fox指出胎盘贫血性梗塞多合并子宫胎盘输入动脉的病变。梗塞胎盘下方的螺旋动脉缺乏正常妊娠的生理变化,且管腔变窄。正常妊娠,与绒毛间隙相通的螺旋动脉,失去正常肌肉及弹力结构,代之以嵌有滋养细胞的类纤维蛋白物质。管腔呈进行性扩张。 Campell(1974)指出高血压、子痫前期、产前出血、糖尿病及慢性肾炎的孕妇约30～35%并发IUGR。以往分娩低体重儿的孕妇,可能有临床查不出的潜在性血管疾病。Bonnar(1976)描述5例血压正常合并IUGR的孕妇,其胎盘均有广泛梗塞,蜕膜曾有闭塞性动脉粥样硬化。Brosens等(1977)研究68例IUGR的胎盘床的活检,在18
It is known that intrauterine growth retardation (IUGR) is associated with placental obstruction. Fox pointed out that the placenta anemia infarction combined with uterine placental afferent artery lesions. The spiral arteries below the infarcted placenta lack physiological changes in normal pregnancy and the lumen is narrowed. Normal pregnancy, and villus interlinked spiral arteries, loss of normal muscle and elastic structure, replaced by trophoblast-embedded fibrin material. The lumen was progressive expansion. Campell (1974) pointed out that about 30-35% of pregnant women with hypertension, preeclampsia, prenatal bleeding, diabetes and chronic glomerulonephritis are complicated by IUGR. In the past pregnant women with low birth weight infants, there may be clinically not found potential vascular disease. Bonnar (1976) described 5 cases of normotensive women with IUGR who had extensive placental infarction and had had decidual atherosclerosis in the decidua. Brosens et al. (1977) studied the biopsy of 68 IUGR placental beds at 18