论文部分内容阅读
我院自1996～1998年收治的76例低血钙症、维生素D缺乏手足搐搦症患儿中,9例同时存在脑发育不良。反复的低钙惊厥导致血压升高,脑耗氧量异常增加。脑发育不良导致脑缺氧缺血,脑血液减少或暂停,加重脑损伤,脑代偿机制失效,脑血流灌注下降,出现第二次血流重新分布,供应大脑半球血流减少,为保证脑内血流,大脑前、中、后动脉灌注边缘带易受损,更加剧惊厥出现,本组病例于惊厥反复发作后,脑发育不良症状、体征、头颅CT均异常改变。说明低钙惊厥可加剧脑发育不良。故提示临床医师引起充分重视。
In our hospital from 1996 to 1998, 76 cases of hypocalcemia, vitamin D deficiency tetany in children, 9 cases of brain dysplasia. Repeated hypocalcemia convulsions lead to elevated blood pressure, abnormal increase in brain oxygen consumption. Brain hypoplasia leading to cerebral hypoxia and ischemia, cerebral blood flow decreased or pause, aggravating brain injury, failure of brain compensatory mechanism, decreased cerebral blood flow perfusion, redistribution of blood flow for the second time, supply of blood flow to the cerebral hemisphere decreased, Brain blood flow, anterior cerebral artery, middle and posterior arterial perfusion marginal zone easily damaged, more serious convulsions appear in this group of patients after recurrent seizures, brain dysplasia, signs, head CT abnormalities were abnormal. Calcification convulsions can aggravate cerebral dysplasia. It prompts clinicians cause full attention.