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一、肾素-血管紧张素-醛固酮系统的生理醛固酮系由肾上腺皮质外层合成,主要促使远肾小管对钠的重吸收与钾的排出。未孕妇女的醛固酮正常分泌量为50～250微克/日。月经周期中,黄体期分泌量较泸泡期为高。血钠减少,血钾增多,短期给予ACTH能使醛固酮的分泌量增加。血钠增加,血钾减少又可使醛固酮的分泌量下降。人类对醛固酮的生理调节机构为肾素-血管紧张素-醛固酮系统。肾素为肾脏合成的酶,无血管收缩作用,但与血浆中的血管紧张素元(即肾素基质)
First, the renin - angiotensin - aldosterone system of physiological aldosterone by the adrenal cortex outer synthesis, mainly to promote the proximal tubular sodium reabsorption and potassium excretion. The normal secretion of aldosterone in pregnant women is 50 ~ 250 micrograms / day. Menstrual cycle, the secretion of luteal phase compared with the high stage of the bubble. Reduced serum sodium, potassium increased short-term administration of ACTH can increase the secretion of aldosterone. Increased serum sodium, serum potassium can reduce aldosterone secretion decreased. Humans human aldosterone physiological regulatory mechanism of renin - angiotensin - aldosterone system. Renin is an enzyme synthesized by the kidneys and has no vasoconstrictor effect but is not associated with angiotensin (plasma renin)